Fluorescent in situ hybridization Reagents

Chemical reagents and products designed to conduct fluorescent in situ hybridizations, a technique used to bind fluorescent probes to specific nucleic acids. Products may be available as kits.

886 – 900 of 1,046 results

886

Advanced Cell Diagnostics 2.5 LS PROBE HS-GPR34-C2

NC3861221 2.5 LS PROBE HS-GPR34-C2

Pricing & Availability

887

Biotium TMRE, 2 mM solution in DMSO

TMRE (Tetramethylrhodamine ethyl ester, perchlorate) and TMRM are preferred dyes for quantitative measurements of membrane potentials using the Nernst equation. TMRE has been used to separate viable and non-apoptotic cells from cell sorting samples. TMRE staining is reversible and does not affect cell proliferation and viability. TMRE is also available in solid form.Properties: Ex/Em (MeOH) = 549/574 nm; Store the solution at 4°C and protect from light; C26H27ClIN2O7; MWt: 515; [115532-52-0];

Pricing & Availability

888

Advanced Cell Diagnostics RNASCOPE PROBE - HS-GIPR-C2

NC3800881 RNASCOPE PROBE - HS-GIPR-C2

Pricing & Availability

889

Advanced Cell Diagnostics RNASCOPE PROBE - HS-GLP1R-CDS

NC3800882 RNASCOPE PROBE - HS-GLP1R-CDS

Pricing & Availability

890

ABclonal Technology [KO Validated] CDKN2A/p16INK4a Rabbit pAb

This gene produces multiple transcript variants differing in their first exons. At least three variants encode distinct proteins; two yield related isoforms that inhibit CDK4 kinase, while a third uses an alternate exon 20 Kb upstream and encodes a structurally unrelated protein from an alternate reading frame (ARF). The ARF product stabilizes the tumor suppressor p53 by binding and sequestering MDM2, an E3 ligase that targets p53 for degradation. Despite structural differences, both CDK inhibitors and ARF regulate cell cycle G1 progression via CDK4 and p53 pathways. This gene is often mutated or deleted in many tumors and acts as a key tumor suppressor.